Activation of necroptosis pathway in podocyte contributes to the pathogenesis of focal segmental glomerular sclerosis
Author:
Funder
Dongguan Science and Technology of Social Development Program
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Nephrology,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s10157-022-02258-1.pdf
Reference28 articles.
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2. Fogo AB. Causes and pathogenesis of focal segmental glomerulosclerosis. Nat Rev Nephrol. 2015;11(2):76–87. https://doi.org/10.1038/nrneph.2014.216.
3. Bedoui S, Herold MJ, Strasser A. Emerging connectivity of programmed cell death pathways and its physiological implications. Nat Rev Mol Cell Biol. 2020;21(11):678–95. https://doi.org/10.1038/s41580-020-0270-8.
4. Lawlor KE, Khan N, Mildenhall A, Gerlic M, Croker BA, D’Cruz AA, et al. RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL. Nat Commun. 2015;6:6282. https://doi.org/10.1038/ncomms7282.
5. Conos SA, Chen KW, De Nardo D, Hara H, Whitehead L, Nunez G, et al. Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner. Proc Natl Acad Sci U S A. 2017;114(6):E961–9. https://doi.org/10.1073/pnas.1613305114.
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