Circ_0000064 knockdown attenuates high glucose-induced proliferation, inflammation and extracellular matrix deposition of mesangial cells through miR-424-5p-mediated WNT2B inhibition in cell models of diabetic nephropathy
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Nephrology,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s10157-022-02241-w.pdf
Reference30 articles.
1. Dounousi E, et al. Improvements in the management of diabetic nephropathy. Rev Diabet Stud. 2015;12(1–2):119–33.
2. Cooper ME. Diabetes: treating diabetic nephropathy-still an unresolved issue. Nat Rev Endocrinol. 2012;8(9):515–6.
3. Badal SS, Danesh FR. New insights into molecular mechanisms of diabetic kidney disease. Am J Kidney Dis. 2014;63(2 Suppl 2):S63-83.
4. Shaw JE, Sicree RA, Zimmet PZ. Global estimates of the prevalence of diabetes for 2010 and 2030. Diabetes Res Clin Pract. 2010;87(1):4–14.
5. Loganathan TS, et al. Interactions among non-coding RNAs in diabetic nephropathy. Front Pharmacol. 2020;11:191.
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