Multiple Sklerose: Stillstand durch Interventionen
Author:
Publisher
Springer Science and Business Media LLC
Subject
Psychiatry and Mental health,Neurology (clinical),Neurology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00115-022-01358-3.pdf
Reference37 articles.
1. Sharrack B, Saccardi R, Alexander T, Badoglio M, Burman J, Farge D et al (2020) Autologous haematopoietic stem cell transplantation and other cellular therapy in multiple sclerosis and immune-mediated neurological diseases: updated guidelines and recommendations from the EBMT Autoimmune Diseases Working Party (ADWP) and the Joint Accreditation Committee of EBMT and ISCT (JACIE). Bone Marrow Transplant 55(2):283–306. https://doi.org/10.1038/s41409-019-0684-0
2. Kavrochorianou N, Markogiannaki M, Haralambous S (2016) IFN-beta differentially regulates the function of T cell subsets in MS and EAE. Cytokine Growth Factor Rev 30:47–54. https://doi.org/10.1016/j.cytogfr.2016.03.013
3. Stinissen P, Raus J, Zhang J (1997) Autoimmune pathogenesis of multiple sclerosis: role of autoreactive T lymphocytes and new immunotherapeutic strategies. Crit Rev Immunol 17(1):33–75. https://doi.org/10.1615/critrevimmunol.v17.i1.20
4. Martin-Saavedra FM, Gonzalez-Garcia C, Bravo B, Ballester S (2008) Beta interferon restricts the inflammatory potential of CD4+ cells through the boost of the Th2 phenotype, the inhibition of Th17 response and the prevalence of naturally occurring T regulatory cells. Mol Immunol 45(15):4008–4019. https://doi.org/10.1016/j.molimm.2008.06.006
5. Sweeney CM, Lonergan R, Basdeo SA, Kinsella K, Dungan LS, Higgins SC et al (2011) IL-27 mediates the response to IFN-beta therapy in multiple sclerosis patients by inhibiting Th17 cells. Brain Behav Immun 25(6):1170–1181. https://doi.org/10.1016/j.bbi.2011.03.007
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