Maternal allergen exposure as a risk factor for childhood asthma-Reference-Cited by-同舟云学术

Maternal allergen exposure as a risk factor for childhood asthma

Published:2006-01 Issue:1 Volume:6 Page:75-80
ISSN:1529-7322
Container-title:Current Allergy and Asthma Reports
language:en
Short-container-title:Curr Allergy Asthma Rep

Author:

Prescott Susan L.

Publisher

Springer Science and Business Media LLC

Subject

Pulmonary and Respiratory Medicine,Immunology and Allergy,Immunology

Link

http://link.springer.com/content/pdf/10.1007/s11882-006-0014-7.pdf

Reference49 articles.

1. Woodcock A, Lowe LA, Murray CS, et al.: Early life environmental control: effect on symptoms, sensitization, and lung function at age 3 years. NAC Manchester Asthma and Allergy Study Group. Am J Respir Crit Care Med 2004, 170:433–439. Children at high risk for allergic disease (n = 251) were prenatally randomized to stringent environmental control for HDM or no intervention. At 3 years of age children in the active group were more likely to have developed sensitization compared with control subjects (risk ratio, 1.61, 95% CI, 1.02–2.55, P = 0.04). However, lung function (airway resistance) was signi.cantly better in the active group.

2. Miles EA, Warner JA, Jones AC, et al.: Peripheral blood mononuclear cell proliferative responses in the first year of life in babies born to atopic parents. Clin Exp Allergy 1996, 26:780–788.

3. Smillie FI, Elder.eld AJ, Patel F, et al.: Lymphoproliferative responses in cord blood and at one year: no evidence for the effect of in utero exposure to dust mite allergens. Clin Exp Allergy 2001, 31:1194–1204.

4. Prescott S, Macaubas C, Holt B, et al.: Transplacental priming of the human immune system to environmental allergens: universal skewing of initial T-cell responses towards Th-2 cytokine pro.le. J Immunol 1998, 160:4730–4737.

5. van der Velden VH, Laan MP, Baert MR, et al.: Selective development of a strong Th2 cytokine pro.le in high-risk children who develop atopy: risk factors and regulatory role of IFN-gamma, IL-4 and IL-10. Clin Exp Allergy 2001, 31:997–1006.

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