Non-steroidal anti-inflammatory drugs and the augmented lipoxygenase pathway: conceivable impact on joint conditions-Reference-Cited by-同舟云学术

Non-steroidal anti-inflammatory drugs and the augmented lipoxygenase pathway: conceivable impact on joint conditions

Published:1992 Issue: Volume: Page:269-274
ISSN:0923-9405
Container-title:Side-Effects of Anti-Inflammatory Drugs 3
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Author:

Bonta Iván L.,Elliott Graham R.

Publisher

Springer Netherlands

Link

http://link.springer.com/content/pdf/10.1007/978-94-011-2982-4_32.pdf

Reference11 articles.

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2. Docherty J.C., Wilson T.W. Indomethacin increases the formation of lipoxygenase products in calcium ionophore stimulated human neutrophils. Biochem Biophys Res Commun. 1987;2:534–538.

3. Elliott G.R., Tak C., Pellens C., Ben-Efraim S., Bonta I.L. Indomethacin stimulation of macrophage cytostasis against MOPC-315 tumor cells is inhibited by both prostaglandin E2 and nordihydroguaiaretic acid, a lipoxygenase inhibitor. Cancer Immunol Immunother. 1988;27:133–136.

4. Elliott G.R., Lauwen A.P.M., Bonta I.L. Prostaglandin E2 inhibits and indomethacin and aspirin enhance, A23187-stimulated leukotriene B4 synthesis by rat peritoneal macrophages. Br J Pharmacol. 1989;96:265–270.

5. Rang H.P., Dale M.M.. Drugs used to suppress inflammatory and immune reactions. In: Rang H.P., Dale M.M., eds. Pharmacology. Edinburgh: Churchill Livingstone; 1987:204–224.

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