1. Hasler, F., Bluestein, H.G., Zwaifler, N.J. and Epstein, L.B. (1983). Analysis of the defects responsible for the impaired regulation of EBV-induced B-cell proliferation by rheumatoid arthritis lymphocytes. II. Role of monocytes and the increased sensitivity of rheumatoid arthritis lymphocytes to prostaglandin E. J. Immunol, 131, 768–72
2. Allison, A.C. (1977). Autoimmune diseases: Concepts of pathogenesis and control. In Talal, N. (ed.) Autoimmunity: Genetic, Virologie, and Clinical Aspects. pp. 92–139. (New York, London: Academic Press)
3. Schwartz, R.S. and Rose, N.R. (eds.) (1986). Autoimmunity: experimental and clinical aspects. Ann. NY. Acad. Sci., 475, 1–427
4. Budd, R.C., Cerottini, J.-C., Horvath, C., Bron, C., Pedrazzini, T., Howe, R.C. and MacDonald, H.R. (1987). Distinction of virgin and memory T lymphocytes. J. Immunol., 138, 3120–9
5. Klickstein, L.B., Shapleigh, C. and Goetzl, E.J. (1980). Lipoxygenation of arachidonic acid as a source of polymorphonuclear chemotactic factors in synovial fluid and tissue in rheumatoid arthritis and spondyloarthritis. J. Clin. Invest., 66, 1166–70