Recent progress on the role of cellular communication network factors (CCN) 3, 4 and 6 in regulating adiposity, liver fibrosis and pancreatic islets
Author:
Funder
NSERC
CIHR
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
https://link.springer.com/content/pdf/10.1007/s12079-023-00765-8.pdf
Reference47 articles.
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2. Alami T, Liu JL (2021) Metabolic Effects of CCN5/WISP2 Gene Deficiency and transgenic overexpression in mice. Int J Mol Sci 22(24):13418
3. Barchetta I, Cimini FA, Capoccia D, De Gioannis R, Porzia A, Mainiero F, Di Martino M, Bertoccini L, De Bernardinis M, Leonetti F, Baroni MG, Lenzi A, Cavallo MG (2017) WISP1 is a marker of systemic and adipose tissue inflammation in dysmetabolic subjects with or without type 2 diabetes. J Endocr Soc 1(6):660–670
4. Borkham-Kamphorst E, Huss S, Van de Leur E, Haas U, Weiskirchen R (2012) Adenoviral CCN 3/NOV gene transfer fails to mitigate liver fibrosis in an experimental bile duct ligation model because of hepatocyte apoptosis. Liver International 32(9):1342–1353
5. Borkham-Kamphorst E, Schaffrath C, Van de Leur E, Haas U, Tihaa L, Meurer SK, Nevzorova YA, Liedtke C, Weiskirchen R (2014) The anti-fibrotic effects of CCN1/CYR61 in primary portal myofibroblasts are mediated through induction of reactive oxygen species resulting in cellular senescence, apoptosis and attenuated TGF-beta signaling. Biochim Biophys Acta 1843(5):902–914
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