NQO1 Bioactivatable Drugs Enhance Radiation Responses-Reference-Cited by-同舟云学术

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NQO1 Bioactivatable Drugs Enhance Radiation Responses

Published:2016 Issue: Volume: Page:225-252
ISSN:
Container-title:Strategies to Enhance the Therapeutic Ratio of Radiation as a Cancer Treatment
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Author:

Bey Erik A.,Meade Julia C.,Silvers Molly A.,Motea Edward A.,Patidar Praveen L.,Brekken Rolf,Deja Stanislaw,Merritt Matthew E.,Kilgore Jessica A.,Liu Yuliang,Huang Xiumei,Li Longshan,Yordy John,Williams Noelle S.,Gao Jinming,Boothman David A.

Publisher

Springer International Publishing

Link

http://link.springer.com/content/pdf/10.1007/978-3-319-45594-5_10

Reference191 articles.

1. Hall EJ. Radiobiology for the Radiologist. 4th ed. Philadelphia: J.B.Lippincott Company; 1994. p. 219–21.

2. Hall EJ. Radiation, the two-edged sword: cancer risks at high and low doses. Cancer J. 2000;6:343–50.

3. Boothman DA, Meyers M, Fukunaga N, Lee SW. Isolation of x-ray-inducible transcripts from radioresistant human melanoma cells. Proc Natl Acad Sci U S A. 1993;90:7200–4.

4. Dong Y, et al. Prostate cancer radiosensitization through poly(ADP-Ribose) polymerase-1 hyperactivation. Cancer Res. 2010;70:8088–96.

5. Chakrabarti G, et al. Tumor-selective use of DNA base excision repair inhibition in pancreatic cancer using the NQO1 bioactivatable drug, beta-lapachone. Sci Rep. 2015;5:17066.

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