Syndrome of persisting thyroid stimulating immunoglobulins and growth promotion of goiter combined with low thyroxine and high triiodothyronine serum levels in drug treated Graves’ disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
http://link.springer.com/content/pdf/10.1007/BF03347623.pdf
Reference21 articles.
1. Pinchera A., Liberti P., Martino E., Fenzi G.F., Grasso L., Rovis L., Baschieri L. Effects of antithyroid therapy on the Long-Acting Thyroid Stimulator and the thyroglobulin antibodies. J. Clin. Endocrinol. Metab. 29: 231, 1969.
2. Fenzi G.F., Hashizume K., Roudebush C.P., DeGroot L.J. Changes in thyroid-stimulating immunoglobulins during antithyroid therapy. J. Clin. Endocrinol. Metab. 48: 572, 1979.
3. Kuzuya N., Chiu S.C., Ikeda H., Uchimura H., Ito K., Nagataki S. Correlation between thyroid stimulators and 3,5,3′-triio- dothyronine suppressibility in patients during treatment for hyperthyroidism with thionamide drugs: comparison of assays by thyroid-stimulating and thyrotropin-displacing activities. J. Clin. Endocrinol. Metab. 48: 706, 1979.
4. McGregor A.M., Petersen M.M., McLachlan S.M., Rooke P., Rees-Smith B., Hall R. Carbimazole and the autoimmune response in Graves’s disease. N. Engl. J. Med. 303: 307, 1980.
5. Docter R., Bos G., Visser T.J., Hennemann G. Thyrotropin binding inhibiting immunoglobulins in Graves’ disease before, during and after antithyroid therapy, and its relation to Long Acting Thyroid Stimula tor. Clin. Endocrinol. 12: 143, 1980.
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