Synthesis rate of dopamine: Difference between corpus striatum and limbic system as a possible explanation of variations in reactions to drugs

Author:

And�n Nils-Erik,Grabowska-And�n Maria,Lindgren Silvana,Thornstr�m Ulla

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,General Medicine

Reference30 articles.

1. Andén N-E (1974) Inhibition of the turnover of the brain dopamine after treatment with the gammaaminobutyrate: 2-oxyglutarate transaminase inhibitor aminooxyacetic acid. Naunyn-Schmiedeberg's Arch Pharmacol 283:419?424

2. Andén N-E (1975) Lesions of the nigro-neostriatal dopamine neurons or the bulbo-spinal norepinephrine and 5-hydroxytryptamine neurons in rats: actions of drugs. Pharmacol Ther B 1:371?380

3. Andén N-E (1976a) The interaction of neuroleptic drugs with striatal and limbic dopaminergic mechanisms. In: Sedvall G, Uvnäs B, Zotterman Y (eds) Antipsychotic drugs: pharmacodynamics and pharmacokinetics. Pergamon, Oxford, pp 217?225

4. Andén N-E (1976b) Animal models of brain dopamine function. In: Birkmayer W, Hornykiewicz O (eds) Advances in parkinsonism. Roche, Basel, pp 169?177

5. Andén N-E (1980) Regulation of monoamine synthesis and utilization by receptors. In: Szekeres L (ed) Handbook of experimental pharmacology, vol 54/I. Springer, Berlin Heidelberg New York, pp 429?462

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