Novel Interactions Involving the Mas Receptor Show Potential of the Renin–Angiotensin system in the Regulation of Microglia Activation: Altered Expression in Parkinsonism and Dyskinesia-Reference-Cited by-同舟云学术

Novel Interactions Involving the Mas Receptor Show Potential of the Renin–Angiotensin system in the Regulation of Microglia Activation: Altered Expression in Parkinsonism and Dyskinesia

Published:2021-01-20 Issue:2 Volume:18 Page:998-1016
ISSN:1933-7213
Container-title:Neurotherapeutics
language:en
Short-container-title:Neurotherapeutics

Author:

Rivas-Santisteban Rafael,Lillo Jaume,Muñoz Ana,Rodríguez-Pérez Ana I.,Labandeira-García José Luís,Navarro Gemma,Franco Rafael^ORCID

Funder

Ministerio de Ciencia, Innovación y Universidades

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Neurology (clinical),Pharmacology

Link

https://link.springer.com/content/pdf/10.1007/s13311-020-00986-4.pdf

Reference89 articles.

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2. Souza LL, Duchene J, Todiras M, Azevedo LCP, Costa-Neto CM, Alenina N, et al. Receptor mas protects mice against hypothermia and mortality induced by endotoxemia. Shock. 2014;41(4):331–6.

3. Santos RAS, Simoes e Silva AC, Maric C, Silva DMR, Machado RP, de Buhr I, et al. Angiotensin-(1-7) is an endogenous ligand for the G protein-coupled receptor Mas. Proc Natl Acad Sci U S A [Internet]. 2003;100(14):8258–63. Available from: http://www.pnas.org/content/100/14/8258.full

4. Villela D, Leonhardt J, Patel N, Joseph J, Kirsch S, Hallberg A, et al. Angiotensin type 2 receptor (AT 2 R) and receptor Mas: a complex liaison. Clin Sci [Internet]. 2015;128(4):227–34. Available from: http://www.clinsci.org/content/128/4/227.abstract%5Cn, http://clinsci.org/lookup/10.1042/CS20130515

5. De Carvalho Santuchi M, Dutra MF, Vago JP, Lima KM, Galvão I, De Souza-Neto FP, et al. Angiotensin-(1-7) and Alamandine Promote Anti-inflammatory Response in Macrophages in Vitro and in Vivo. Mediators Inflamm [Internet]. 2019 [cited 2020 Apr 29];2019(1):2401081. Available from: https://doi.org/10.1155/2019/2401081

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