Consequences of Metabolic Disruption in Alzheimer’s Disease Pathology-Reference-Cited by-同舟云学术

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Consequences of Metabolic Disruption in Alzheimer’s Disease Pathology

Published:2019-07 Issue:3 Volume:16 Page:600-610
ISSN:1933-7213
Container-title:Neurotherapeutics
language:en
Short-container-title:Neurotherapeutics

Author:

Ryu J. C.,Zimmer E. R.,Rosa-Neto P.,Yoon S. O.

Funder

National Institute on Aging

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Neurology (clinical),Pharmacology

Link

http://link.springer.com/content/pdf/10.1007/s13311-019-00755-y.pdf

Reference147 articles.

1. Allen KV, Frier BM, Strachan MW. 2004. The relationship between type 2 diabetes and cognitive dysfunction: longitudinal studies and their methodological limitations. Eur J Pharmacol 490: 169–175

2. Amatniek JC, Hauser WA, DelCastillo-Castaneda C, Jacobs DM, Marder K, et al. 2006. Incidence and predictors of seizures in patients with Alzheimer’s disease. Epilepsia 47: 867–872

3. Asher G, Gatfield D, Stratmann M, Reinke H, Dibner C, et al. 2008. SIRT1 regulates circadian clock gene expression through PER2 deacetylation. Cell 134: 317–328

4. Baker LD, Cross DJ, Minoshima S, Belongia D, Watson GS, Craft S. 2011. Insulin resistance and Alzheimer-like reductions in regional cerebral glucose metabolism for cognitively normal adults with prediabetes or early type 2 diabetes. Arch Neurol 68: 51–57

5. Balland E, Dam J, Langlet F, Caron E, Steculorum S, et al. 2014. Hypothalamic tanycytes are an ERK-gated conduit for leptin into the brain. Cell Metab 19: 293–301

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