TBC1 domain family member 25 protects against myocardial apoptosis and the proinflammatory response triggered by ischemia–reperfusion injury through suppression of the TAK1-JNK/p38 MAPK signaling cascade
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Developmental Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11626-023-00826-7.pdf
Reference27 articles.
1. Aashaq S, Batool A, Andrabi KI (2019) TAK1 mediates convergence of cellular signals for death and survival. Apoptosis 24:3–20
2. Chen C, Yu LT, Cheng BR, Xu JL, Cai Y, Jin JL, Feng RL, Xie L, Qu XY, Li D, Liu J, Li Y, Cui XY, Lu JJ, Zhou K, Lin Q, Wan J (2021a) Promising therapeutic candidate for myocardial ischemia/reperfusion injury: what are the possible mechanisms and roles of phytochemicals? Front Cardiovasc Med 8:792592
3. Chen SY, Zhang HP, Li J, Shi JH, Tang HW, Zhang Y, Zhang JK, Wen PH, Wang ZH, Shi XY, He YT, Hu BW, Yang H, Guo WZ, Zhang SJ (2021b) Tripartite motif-containing 27 attenuates liver ischemia/reperfusion injury by suppressing transforming growth factor beta-activated kinase 1 (TAK1) by TAK1 binding protein 2/3 degradation. Hepatology 73:738–758
4. Dong L, Shen Z, Chi H, Wang Y, Shi Z, Fang H, Yang Y, Rong J (2023) Research progress of Chinese medicine in the treatment of myocardial ischemia-reperfusion injury. Am J Chin Med 51:1–17
5. Gabernet-Castello C, O’Reilly AJ, Dacks JB, Field MC (2013) Evolution of Tre-2/Bub2/Cdc16 (TBC) Rab GTPase-activating proteins. Mol Biol Cell 24:1574–1583
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