Hace1 overexpression mitigates myocardial hypoxia/reoxygenation injury via the effects on Keap1/Nrf2 pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Developmental Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11626-022-00725-3.pdf
Reference33 articles.
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2. Bai B, Zeng G, Chen R, Ai Y, Qiang H (2021) Upregulation of iASPP ameliorates hypoxia/reoxygenation-induced apoptosis and oxidative stress in cardiomyocytes by upregulating Nrf2 signaling. J Biochem Mol Toxicol 35:e22686
3. Barancik M, Gresova L, Bartekova M, Dovinova I (2016) Nrf2 as a key player of redox regulation in cardiovascular diseases. Physiol Res 65 Suppl 1:S1–S10
4. Bugger H, Pfeil K (2020) Mitochondrial ROS in myocardial ischemia reperfusion and remodeling. Biochim Biophys Acta Mol Basis Dis 1866:165768
5. Collaborators GBDCoD (2018) Global, regional, and national age-sex-specific mortality for 282 causes of death in 195 countries and territories, 1980–2017: a systematic analysis for the Global Burden of Disease Study 2017. Lancet 392:1736–1788
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1. HACE1 exerts a neuroprotective role against oxidative stress in cerebral ischemia–reperfusion injury by activating the PI3K/AKT/Nrf2 pathway;Neuroscience;2024-11
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