Reduction of myocardial leukocyte accumulation and myocardial infarct size following administration of BAY u3405, a thromboxane A2 receptor antagonist, in myocardial ischaemia-reperfusion injury-Reference-Cited by-同舟云学术

Reduction of myocardial leukocyte accumulation and myocardial infarct size following administration of BAY u3405, a thromboxane A2 receptor antagonist, in myocardial ischaemia-reperfusion injury

Published:1993-07 Issue:3-4 Volume:39 Page:143-149
ISSN:0065-4299
Container-title:Agents and Actions
language:en
Short-container-title:Agents and Actions

Author:

Squadrito F.,Ioculano M.,Altavilla D.,Zingarelli B.,Canale P.,Campo G. M.,Saitta A.,Oriti S.,Faggiotto A.,Caputi A. P.

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Toxicology,Pharmacology,Immunology

Link

http://link.springer.com/content/pdf/10.1007/BF01998967.pdf

Reference29 articles.

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2. A. M. Lefer,Significance of lipid mediators in shock states. Circ. Shock27, 3–12 (1989).

3. P. Walinsky, J. B. Smith and A. M. Lefer et al.,Thromboxane A 2 in acute myocardial infarction. Am. Heart J.108, 868–872 (1984).

4. S. Morooka, M. Kobayashi and T. Shimamoto,Experimental ischemic heart disease induced by thromboxane A 2 in rabbits. Jap. Circ. J.41, 1373–1379 (1978).

5. E. F. Smith III, A. M. Lefer and D. Aharony et al.,Carbocyclic thromboxane A 2 : Aggravation of myocardial ischemia by a new synthetic thromboxane analog. Prostaglandins21, 443–456 (1981).

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