The effects of acute acetaminophen toxicity on hepatic mRNA expression of SOD, CAT, GSH-Px, and levels of peroxynitrite, nitric oxide, reduced glutathione, and malondialdehyde in rabbit-Reference-Cited by-同舟云学术

The effects of acute acetaminophen toxicity on hepatic mRNA expression of SOD, CAT, GSH-Px, and levels of peroxynitrite, nitric oxide, reduced glutathione, and malondialdehyde in rabbit

Published:2008-11-28 Issue:1-2 Volume:323 Page:31-38
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Cigremis Yilmaz,Turel Huseyin,Adiguzel Kevser,Akgoz Muslum,Kart Asim,Karaman Musa,Ozen Hasan

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11010-008-9961-8.pdf

Reference35 articles.

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2. Sheen CL, Dillon JF, Bateman DN et al (2002) Paracetamol toxicity: epidemiology, prevention and costs to the health-care system. QJM 95:609–619. doi: 10.1093/qjmed/95.9.609

3. Dahlin DC, Miwa GT, Lu AYH et al (1984) N-Acetyl-para-benzoquinone imine—a cytochrome-P-450-mediated oxidation-product of acetaminophen. Proc Natl Acad Sci USA 81:1327–1331

4. Nelson SD (1990) Molecular mechanisms of the hepatotoxicity caused by acetaminophen. Semin Liver Dis 10:267–278

5. Mitchell JR, Jollow DJ, Potter WZ et al (1973) Acetaminophen-induced hepatic necrosis. 4. Protective role of glutathione. J Pharmacol Exp Ther 187:211–217

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