Oxidative stress does not play a primary role in the toxicity induced with clinical doses of doxorubicin in myocardial H9c2 cells-Reference-Cited by-同舟云学术

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Oxidative stress does not play a primary role in the toxicity induced with clinical doses of doxorubicin in myocardial H9c2 cells

Published:2016-01-30 Issue:1-2 Volume:413 Page:199-215
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Rharass Tareck,Gbankoto Adam,Canal Christophe,Kurşunluoğlu Gizem,Bijoux Amandine,Panáková Daniela,Ribou Anne-Cécile

Funder

Ligue Nationale Contre le Cancer

ERASMUS program

Helmholtz Young Investigator Program

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11010-016-2653-x.pdf

Reference70 articles.

1. Minotti G, Menna P, Salvatorelli E, Cairo G, Gianni L (2010) Anthracyclines: molecular advances and pharmacologic developments in antitumor activity and cardiotoxicity. Pharmacol Rev 56:185–229

2. Pommier Y, Leo E, Zhang H, Marchand C (2010) DNA topoisomerases and their poisoning by anticancer and antibacterial drugs. Chem Biol 17:421–433

3. Cutts SM, Nudelman A, Rephaeli A, Phillips DR (2005) The power and potential of doxorubicin-DNA adducts. IUBMB Life 57:73–81

4. Jung K, Reszka R (2001) Mitochondria as subcellular targets for clinically useful anthracyclines. Adv Drug Deliv Rev 49:87–105

5. Octavia Y, Tocchetti CG, Gabrielson KL, Janssens S, Crijns H, Moens AL (2012) Doxorubicin-induced cardiomyopathy: from molecular mechanisms to therapeutic strategies. J Mol Cell Cardiol 52:1213–1225

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