Pleiotropic, non-cell death-associated effects of inhibitors of receptor-interacting protein kinase 1 in the heart
Author:
Funder
APVV
VEGA,
VEGA
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11010-021-04136-y.pdf
Reference45 articles.
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2. Stanger BZ, Leder P, Lee T-H et al (1995) RIP: a novel protein containing a death domain that interacts with Fas/APO-1 (CD95) in yeast and causes cell death. Cell 81:513–523. https://doi.org/10.1016/0092-8674(95)90072-1
3. Kang T-B, Jeong J-S, Yang S-H et al (2018) Caspase-8 deficiency in mouse embryos triggers chronic RIPK1-dependent activation of inflammatory genes, independently of RIPK3. Cell Death Differ 25:1107–1117. https://doi.org/10.1038/s41418-018-0104-9
4. O’Donnell MA, Legarda-Addison D, Skountzos P et al (2007) Ubiquitination of RIP1 regulates an NF-κB-independent cell death switch in TNF signaling. Curr Biol 17:418–424. https://doi.org/10.1016/j.cub.2007.01.027
5. Kelliher MA, Grimm S, Ishida Y et al (1998) The death domain kinase RIP mediates the TNF-induced NF-κB signal. Immunity 8:297–303. https://doi.org/10.1016/S1074-7613(00)80535-X
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