CircPDE7B/miR-661 axis accelerates the progression of human keloid fibroblasts by upregulating fibroblast growth factor 2 (FGF2)
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11010-021-04345-5.pdf
Reference34 articles.
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2. Limandjaja GC, Niessen FB, Scheper RJ, Gibbs S (2020) The keloid disorder: heterogeneity, histopathology, mechanisms and models. Front Cell Dev Biol 8:360. https://doi.org/10.3389/fcell.2020.00360
3. He Y, Deng Z, Alghamdi M, Lu L, Fear MW, He L (2017) From genetics to epigenetics: new insights into keloid scarring. Cell Prolif. https://doi.org/10.1111/cpr.12326
4. Marneros AG, Krieg T (2004) Keloids–clinical diagnosis, pathogenesis, and treatment options. J Dtsch Dermatol Ges 2:905–913. https://doi.org/10.1046/j.1439-0353.2004.04077.x
5. Luo S, Benathan M, Raffoul W, Panizzon RG, Egloff DV (2001) Abnormal balance between proliferation and apoptotic cell death in fibroblasts derived from keloid lesions. Plast Reconstr Surg 107:87–96. https://doi.org/10.1097/00006534-200101000-00014
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