Cisplatin treatment reduces contraction to angiotensin II by altering expression of angiotensin II receptors: a pilot study-Reference-Cited by-同舟云学术

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Cisplatin treatment reduces contraction to angiotensin II by altering expression of angiotensin II receptors: a pilot study

Published:2023-04-01 Issue:12 Volume:478 Page:2907-2916
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

McSweeney Kristen Renee,Gadanec Laura Kate,Kubatka Peter,Caprnda Martin,Gaspar Ludovit,Prosecky Robert,Delev Delian,Kruzliak Peter,Apostolopoulos Vasso,Zulli Anthony

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s11010-023-04706-2.pdf

Reference39 articles.

1. Galluzzi L, Senovilla L, Vitale I, Michels J, Martins I, Kepp O, Castedo M, Kroemer G (2012) Molecular mechanisms of cisplatin resistance. Oncogene 31:1869–1883

2. Khadrawy YA, Hosny EN, El-Gizawy MM, Sawie HG, Aboul Ezz HS (2021) The effect of curcumin nanoparticles on cisplatin-induced cardiotoxicity in male Wistar albino rats. Cardiovasc Toxicol 21:433–443

3. McSweeney KR, Gadanec LK, Qaradakhi T, Ali BA, Zulli A, Apostolopoulos V (2021) Mechanisms of cisplatin-induced acute kidney injury: pathological mechanisms, pharmacological interventions, and genetic mitigations. Cancers 13:1572

4. Ramesh G, Reeves WB (2002) TNF-α mediates chemokine and cytokine expression and renal injury in cisplatin nephrotoxicity. J Clin Investig 110:835–842

5. Zhang B, Ramesh G, Norbury C, Reeves W (2007) Cisplatin-induced nephrotoxicity is mediated by tumor necrosis factor-α produced by renal parenchymal cells. Kidney Int 72:37–44

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