Specific MAPK inhibitors prevent hyperglycemia-induced renal diseases in type 1 diabetic mouse model-Reference-Cited by-同舟云学术

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Specific MAPK inhibitors prevent hyperglycemia-induced renal diseases in type 1 diabetic mouse model

Published:2016-07-07 Issue:1-2 Volume:419 Page:1-9
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Hong Zhe,Hong Zongyuan,Wu Denglong,Nie Hezhongrong

Funder

the National Natural Science Foundation of China

Anhui Provincial Programs for Science and Technology Development

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11010-016-2722-1.pdf

Reference35 articles.

1. Brownlee M (2001) Biochemistry and molecular cell biology of diabetic complications. Nature 414:813–820. doi: 10.1038/414813a

2. Alique M, Civantos E, Sanchez-Lopez E, Lavoz C, Rayego-Mateos S, Rodrigues-Diez R, Garcia-Redondo AB, Egido J, Ortiz A, Rodriguez-Puyol D, Rodriguez-Puyol M, Ruiz-Ortega M (2014) Integrin-linked kinase plays a key role in the regulation of angiotensin II-induced renal inflammation. Clin Sci (Lond) 127:19–31. doi: 10.1042/CS20130412

3. Vaziri ND (2004) Roles of oxidative stress and antioxidant therapy in chronic kidney disease and hypertension. Curr Opin Nephrol Hypertens 13:93–99

4. Carey RM, Siragy HM (2003) The intrarenal renin-angiotensin system and diabetic nephropathy. Trends Endocrinol Metab 14:274–281

5. Kagami S, Border WA, Miller DE, Noble NA (1994) Angiotensin II stimulates extracellular matrix protein synthesis through induction of transforming growth factor-beta expression in rat glomerular mesangial cells. J Clin Invest 93:2431–2437. doi: 10.1172/JCI117251

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