Tumor necrosis factor α stimulates endogenous apolipoprotein A-I expression and secretion by human monocytes and macrophages: role of MAP-kinases, NF-κB, and nuclear receptors PPARα and LXRs-Reference-Cited by-同舟云学术

Tumor necrosis factor α stimulates endogenous apolipoprotein A-I expression and secretion by human monocytes and macrophages: role of MAP-kinases, NF-κB, and nuclear receptors PPARα and LXRs

Published:2018-02-13 Issue:1-2 Volume:448 Page:211-223
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Shavva Vladimir S.,Mogilenko Denis A.,Nekrasova Ekaterina V.,Trulioff Andrey S.,Kudriavtsev Igor V.,Larionova Ekaterina E.,Babina Anna V.,Dizhe Ella B.,Missyul Boris V.,Orlov Sergey V.^ORCID

Funder

Russian Science Foundation

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/article/10.1007/s11010-018-3327-7/fulltext.html

Reference54 articles.

1. Lewis GF, Rader DJ (2005) New insights into the regulation of HDL metabolism and reverse cholesterol transport. Circ Res 96:1221–1232

2. Maciejko JJ, Holmes DR, Kottke BA, Zinsmeister AR, Dinh DM, Mao SJ (1983) Apolipoprotein A-I as a marker of angiographically assessed coronary-artery disease. N Engl J Med 309:385–389

3. Schaefer EJ, Heaton WH, Wetzel MG, Brewer HB (1982) Plasma apolipoprotein A-1 absence associated with a marked reduction of high density lipoproteins and premature coronary artery disease. Arteriosclerosis 2:16–26