Epsilon-aminocaproic acid prevents high glucose and insulin induced-invasiveness in MDA-MB-231 breast cancer cells, modulating the plasminogen activator system-Reference-Cited by-同舟云学术

Epsilon-aminocaproic acid prevents high glucose and insulin induced-invasiveness in MDA-MB-231 breast cancer cells, modulating the plasminogen activator system

Published:2017-06-13 Issue:1-2 Volume:437 Page:65-80
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Viedma-Rodríguez Rubí,Martínez-Hernández María Guadalupe,Flores-López Luis Antonio,Baiza-Gutman Luis Arturo

Funder

Programa de Apoyo a Proyectos de Investigación e Innovación Tecnológica (PAPIIT), Dirección General de Asuntos del Personal Académico (DGAPA), Universidad Nacional Autónoma de México (UNAM)

Posdoctoral Fellowship, Dirección General de Asuntos del Personal Académico (DGAPA), Universidad Nacional Autónoma de México (UNAM).

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/article/10.1007/s11010-017-3096-8/fulltext.html

Reference80 articles.

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2. Takatani-Nakase T, Matsui C, Maeda S, Kawahara S, Takahashi K (2014) High glucose level promotes migration behavior of breast cancer cells through zinc and its transporters. PLoS ONE 9:e90136. doi: 10.1371/journal.pone.0090136

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4. Zhu S, Yao F, Li WH, Wan JN, Zhang YM, Tang Z, Khan S, Wang CH, Sun SR (2013) PKC?-dependent activation of the ubiquitin proteasome system is responsible for high glucose-induced human breast cancer MCF-7 cell proliferation, migration and invasion. Asian Pac J Cancer Prev 14:5687–5692

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