Somatostatin activates Ras and ERK1/2 via a G protein βγ-subunit-initiated pathway in thyroid cells-Reference-Cited by-同舟云学术

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Somatostatin activates Ras and ERK1/2 via a G protein βγ-subunit-initiated pathway in thyroid cells

Published:2015-10-16 Issue:1-2 Volume:411 Page:253-260
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Rodríguez-Álvarez Francisco J.,Jiménez-Mora Eva,Caballero María,Gallego Beatriz,Chiloeches Antonio,Toro Mª José

Funder

Ministerio de Ciencia e Innovación

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11010-015-2587-8.pdf

Reference32 articles.

1. Medina DL, Toro MJ, Santisteban P (2000) Somatostatin interferes with thyrotropin-induced G1-S transition mediated by cAMP-dependent protein kinase and phosphatidylinositol 3-kinase. Involvement of RhoA and cyclin E cyclin-dependent kinase 2 complexes. J Biol Chem 275:15549–15556

2. Riesco-Eizaguirre G, Santisteban P (2006) A perspective view of sodium iodide symporter research and its clinical implications. Eur J Endocrinol 155:495–512

3. Patel YC (1999) Somatostatin and its receptor family. Front Neuroendocrinol 20:157–198

4. Medina DL, Velasco JA, Santisteban P (1999) Somatostatin is expressed in FRTL-5 Thyroid cells and prevents thyrotropin-mediated down-regulation of the cyclin-dependent kinase inhibitor p27kip1. Endocrinology 140:87–95

5. Birnbaumer L (2007) Expansion of signal transduction by G proteins. The second 15 years or so: from 3 to 16 alpha subunits plus betagamma dimers. Biochim Biophys Acta 1768:772–793

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