Protocol to Study West Nile Virus Infection in Brain Slices In Vitro
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Publisher
Springer US
Link
https://link.springer.com/content/pdf/10.1007/978-1-0716-2760-0_10
Reference5 articles.
1. Petzold A, Groves M, Leis AA, Scaravilli F et al (2010) Neuronal and glial cerebrospinal fluid protein biomarkers are elevated after West Nile virus infection. Muscle Nerve 41:42–49. https://doi.org/10.1002/mus.21448
2. Blakely PK, Kleinschmidt-DeMasters BK, Tyler KL, Irani DN (2009) Disrupted glutamate transporter expression in the spinal cord with acute flaccid paralysis caused by West Nile virus infection. J Neuropathol Exp Neurol 68:1061–1072. https://doi.org/10.1097/NEN.0b013e3181b8ba14
3. Leis AA, Stokic DS, Petzold A (2012) Glial S100B is elevated in serum across the spectrum of West Nile virus infection. Muscle Nerve 45:826–830. https://doi.org/10.1002/mus.23241
4. Quick ED, Leser JS, Clarke P, Tyler KL (2014) Activation of intrinsic immune responses and microglial phagocytosis in an ex vivo spinal cord slice culture model of West Nile virus infection. J Virol 88:13005–13014. https://doi.org/10.1128/JVI.01994-14
5. Clarke P, Leser JS, Quick ED, Dionne KR et al (2014) Death receptor-mediated apoptotic signaling is activated in the brain following infection with West Nile virus in the absence of a peripheral immune response. J Virol 88:1080–1089. https://doi.org/10.1128/JVI.02944-13
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