In Vivo Evaluation of Exon 51 Skipping in hDMD/Dmd-null Mice
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Publisher
Springer US
Link
https://link.springer.com/content/pdf/10.1007/978-1-0716-3036-5_23
Reference26 articles.
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2. Hoffman EP, Brown RH, Kunkel LM (1987) Dystrophin: the protein product of the Duchenne muscular dystrophy locus. Cell 51:919–928. https://doi.org/10.1016/0092-8674(87)90579-4
3. Cox GA, Cole NM, Matsumura K et al (1993) Overexpression of dystrophin in transgenic mdx mice eliminates dystrophic symptoms without toxicity. Nature 364:725–729. https://doi.org/10.1038/364725a0
4. Koenig M, Beggs AH, Moyer M et al (1989) The molecular basis for Duchenne versus Becker muscular dystrophy: correlation of severity with type of deletion. Am J Hum Genet 45:498–506
5. Monaco AP, Bertelson CJ, Liechti-Gallati S et al (1988) An explanation for the phenotypic differences between patients bearing partial deletions of the DMD locus. Genomics 2:90–95. https://doi.org/10.1016/0888-7543(88)90113-9
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