DNA double-strand breaks: a potential therapeutic target for neurodegenerative diseases
Author:
Funder
Department of Defense
William and Ella Owens Medical Research Foundation
National Institutes of Health
Publisher
Springer Science and Business Media LLC
Subject
Genetics
Link
http://link.springer.com/content/pdf/10.1007/s10577-019-09617-x.pdf
Reference123 articles.
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2. Ambrosio S, Di Palo G, Napolitano G et al (2016) Cell cycle-dependent resolution of DNA double-strand breaks. Oncotarget 7:4949–4960. https://doi.org/10.18632/oncotarget.6644
3. Babić Leko M, Župunski V, Kirincich J et al (2019) Molecular Mechanisms of neurodegeneration related to C9orf72 hexanucleotide repeat expansion. Behav Neurol 2019:2909168. https://doi.org/10.1155/2019/2909168
4. Barzilai A, Biton S, Shiloh Y (2008) The role of the DNA damage response in neuronal development, organization and maintenance. DNA Repair 7:1010–1027. https://doi.org/10.1016/j.dnarep.2008.03.005
5. Becker LA, Huang B, Bieri G et al (2017) Therapeutic reduction of ataxin-2 extends lifespan and reduces pathology in TDP-43 mice. Nature 544:367–371. https://doi.org/10.1038/nature22038
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