Restriction ofβ-adrenergic responsiveness in hypertrophied ventricular myocardium of rats

Author:

Hein B.,Janke J.

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Reference13 articles.

1. Balazs, T.: Cardiotoxicity of isoproterenol in experimental animals. Influence of stress, obesity, and repeated dosing. In: Recent Advances in Studies on Cardiac Structure and Metabolism, Vol. 1, p. 770–778. Myocardiology, (E. Bajusz andG. Rona, eds.) (Baltimore 1972).

2. Fleckenstein, A.: Specific inhibitors and promoters of calcium action in the excitation-contraction coupling of heart muscle and their role in the prevention or production of myocardial lesions. In: Calcium and the Heart, p. 135–188 (P. Harris andL. Opie, eds.), (London-New York 1971).

3. Fleckenstein, A., J. Janke, H. J. Döring, O. Leder: Key role of Ca in the production of noncoronarogenic myocardial necroses. In: Recent Advances in Studies on Cardiac Structure and Metabolism, Vol. 6, p. 21–32. Pathophysiology and Morphology of Myocardial Cell Alterations (A. Fleckenstein andG. Rona, eds.), (Baltimore 1975).

4. Hein, B., O. Pachinger, J. Janke, A. Fleckenstein: Changes in isoproterenol-induced transmembrane Ca uptake and high-energy phosphate content of the hypertrophied ventricular myocardium of rats. In: Les Surcharges Cardiaques, Collogue INSERM, p. 205–208. (P. Y. Hatt, ed.) (Paris 1972).

5. Hein, B., A. Fleckenstein, J. Janke, K. Eckardt, H. J. Ludin: Einschränkung der Isoproterenol-induzierten45Ca++-Netto-Aufnahme und der Utilisation von energiereichem Phosphat im hypertrophierten Rattenmyokard. Verh. Dtsch. Ges. Kreislaufforschg.38, 196 (1972).

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