Study of Common Genetic Variant S447X in Lipoprotein Lipase and Its Association with Lipids and Lipoproteins in Type 2 Diabetic Patients
Author:
Publisher
Springer Science and Business Media LLC
Subject
Clinical Biochemistry
Link
http://link.springer.com/content/pdf/10.1007/s12291-015-0531-z.pdf
Reference42 articles.
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2. Hokanson JE, Austin MA. Plasma triglyceride level is a risk factor for cardiovascular disease independent of high-density lipoprotein cholesterol level: a meta-analysis of population-based prospective studies. J Cardiovasc Risk. 1996;3(2):213–9.
3. Krauss RM. Atherogenicity of Triglyceride-Rich Lipoproteins. The American Journal of Cardiology 1998; 81 (4, Supplement 1):13B-17B.
4. Fortson MR, Freedman SN, Webster PD III. Clinical assessment of hyperlipidemic pancreatitis. Am J Gastroenterol. 1995;90(12):2134–9.
5. Olivecrona G, Beisiegel U. Lipid binding of apolipoprotein CII is required for stimulation of lipoprotein lipase activity against apolipoprotein CII-deficient chylomicrons. Arterioscler Thromb Vasc Biol. 1997;17(8):1545–9.
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1. The influence of LPL S447X variants and obesity on lipid profile, oxidative stress, and the risk of T2DM: A case‐control study;Health Science Reports;2023-01
2. Characteristics of the gut microbiome in patients with prediabetes and type 2 diabetes;PeerJ;2021-03-24
3. Associations of the LPL S447X and Hind III Polymorphism with Type 2 Diabetes Mellitus Risk: A Meta-Analysis;Hormone and Metabolic Research;2020-09-04
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