Therapie des klassischen 21‑Hydroxylase-Mangel-Adrenogenitalen Syndroms (AGS) im Kindes- und Jugendalter
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology, Diabetes and Metabolism
Link
http://link.springer.com/content/pdf/10.1007/s41969-019-00083-8.pdf
Reference14 articles.
1. Speiser PW, Arlt W, Auchus RJ, et al (2018) Congenital adrenal hyperplasia due to steroid 21-hydroxylase deficiency: an endocrine society clinical practice guideline. J Clin Endocrinol Metab 103(11):4043–4088
2. Strnadová KA, Votava F, Lebl J, et al. (2007) Prevalence of congenital adrenal hyperplasia among sudden infant death in the Czech Republic and Austria. Eur J Pediatr 166(1):1–4
3. Reisch N, Willige M, Kohn D, et al. (2012) Frequency and causes of adrenalcrises over lifetime in patients with 21-hydroxylase deficiency. Eur J Endocrinol 167(1):35–42
4. Odenwald B, Nennstiel-Ratzel U, Dörr HG, et al. (2016) Children with classic congenital adrenal hyperplasia experience salt loss and hypoglycemia: evaluation of adrenal crises during the first six years of life. Eur J Endocrinol 174(2):177–186
5. Bonfig W, Schmidt H, Schwarz HP (2011) Growth patterns in the first three years of life in children with classical congenital adrenal hyperplasia diagnosed by newborn screening and treated with low doses of hydrocortisone. Horm Res Paediatr 75(1):32–37
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