Reply: Soluble oligomers or insoluble fibrils?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical),Pathology and Forensic Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00401-023-02634-5.pdf
Reference34 articles.
1. Busche MA, Wegmann S, Dujardin S, Commins C, Schiantarelli J, Klickstein N et al (2019) Tau impairs neural circuits, dominating amyloid-beta effects, in Alzheimer models in vivo. Nat Neurosci 22:57–64. https://doi.org/10.1038/s41593-018-0289-8
2. Colom-Cadena M, Davies C, Sirisi S, Lee JE, Simzer EM, Tzioras M et al (2023) Synaptic oligomeric tau in Alzheimer’s disease—a potential culprit in the spread of tau pathology through the brain. Neuron 111:2170-2183 e2176. https://doi.org/10.1016/j.neuron.2023.04.020
3. DeVos SL, Corjuc BT, Oakley DH, Nobuhara CK, Bannon RN, Chase A et al (2018) Synaptic Tau seeding precedes tau pathology in human Alzheimer’s disease brain. Front Neurosci 12:267. https://doi.org/10.3389/fnins.2018.00267
4. Dimou E, Katsinelos T, Meisl G, Tuck BJ, Keeling S, Smith AE et al (2023) Super-resolution imaging unveils the self-replication of tau aggregates upon seeding. Cell Rep 42:112725. https://doi.org/10.1016/j.celrep.2023.112725
5. Dujardin S, Commins C, Lathuiliere A, Beerepoot P, Fernandes AR, Kamath TV et al (2020) Tau molecular diversity contributes to clinical heterogeneity in Alzheimer’s disease. Nat Med 26:1256–1263. https://doi.org/10.1038/s41591-020-0938-9
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