Mutations of CARD11 but not TNFAIP3 may activate the NF-κB pathway in primary CNS lymphoma-Reference-Cited by-同舟云学术

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Mutations of CARD11 but not TNFAIP3 may activate the NF-κB pathway in primary CNS lymphoma

Published:2010-06-11 Issue:4 Volume:120 Page:529-535
ISSN:0001-6322
Container-title:Acta Neuropathologica
language:en
Short-container-title:Acta Neuropathol

Author:

Montesinos-Rongen Manuel,Schmitz Roland,Brunn Anna,Gesk Stefan,Richter Julia,Hong Ke,Wiestler Otmar D.,Siebert Reiner,Küppers Ralf,Deckert Martina

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Pathology and Forensic Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00401-010-0709-7.pdf

Reference29 articles.

1. Boone DL, Turer EE, Lee EG et al (2004) The ubiquitin-modifying enzyme A20 is required for termination of Toll-like receptor responses. Nat Immunol 5:1052–1060

2. Chanudet E, Huang Y, Ichimura K et al (2010) A20 is targeted by promoter methylation, deletion and inactivating mutation in MALT lymphoma. Leukemia 24:483–487

3. Compagno M, Lim WK, Grunn A et al (2009) Mutations of multiple genes cause deregulation of NF-kappaB in diffuse large B-cell lymphoma. Nature 459:717–721

4. Courts C, Montesinos-Rongen M, Martin-Subero JI et al (2007) Transcriptional profiling of the nuclear factor-kappaB pathway identifies a subgroup of primary lymphoma of the central nervous system with low BCL10 expression. J Neuropathol Exp Neurol 66:230–237

5. Davis RE, Brown KD, Siebenlist U, Staudt LM (2001) Constitutive nuclear factor kappaB activity is required for survival of activated B cell-like diffuse large B cell lymphoma cells. J Exp Med 194:1861–1874

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