GluR2/3, NMDA?1 and GABAA receptors in Creutzfeldt-Jakob disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical),Pathology and Forensic Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00401-003-0732-z.pdf
Cited by 19 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Analysis of RNA Expression Profiles Identifies Dysregulated Vesicle Trafficking Pathways in Creutzfeldt-Jakob Disease;Molecular Neurobiology;2018-11-16
2. Inhibition of group-I metabotropic glutamate receptors protects against prion toxicity;PLOS Pathogens;2017-11-27
3. Uncontrolled SFK-mediated protein trafficking in prion and Alzheimer's disease;Prion;2016-09-02
4. Altered Mitochondria, Protein Synthesis Machinery, and Purine Metabolism Are Molecular Contributors to the Pathogenesis of Creutzfeldt–Jakob Disease;Journal of Neuropathology & Experimental Neurology;2016-06-12
5. Dysfunction of Glutamate Receptors in Microglia May Cause Neurodegeneration;Current Alzheimer Research;2016-03-01
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