Misfolded SOD1 is not a primary component of sporadic ALS-Reference-Cited by-同舟云学术

Misfolded SOD1 is not a primary component of sporadic ALS

Published:2017-02-28 Issue:1 Volume:134 Page:97-111
ISSN:0001-6322
Container-title:Acta Neuropathologica
language:en
Short-container-title:Acta Neuropathol

Author:

Da Cruz Sandrine,Bui Anh,Saberi Shahram,Lee Sandra K.,Stauffer Jennifer,McAlonis-Downes Melissa,Schulte Derek,Pizzo Donald P.,Parone Philippe A.,Cleveland Don W.,Ravits John

Funder

Amyotrophic Lateral Sclerosis Association

National Institutes of Health

National Institute of Neurological Disorders and Stroke

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Pathology and Forensic Medicine

Link

http://link.springer.com/article/10.1007/s00401-017-1688-8/fulltext.html

Reference73 articles.

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2. Audet JN, Gowing G, Julien JP (2010) Wild-type human SOD1 overexpression does not accelerate motor neuron disease in mice expressing murine Sod1 G86R. Neurobiol Dis 40:245–250. doi: 10.1016/j.nbd.2010.05.031

3. Ayers JI, Fromholt S, Koch M, DeBosier A, McMahon B, Xu G, Borchelt DR (2014) Experimental transmissibility of mutant SOD1 motor neuron disease. Acta Neuropathol 128:791–803. doi: 10.1007/s00401-014-1342-7

4. Ayers JI, Xu G, Pletnikova O, Troncoso JC, Hart PJ, Borchelt DR (2014) Conformational specificity of the C4F6 SOD1 antibody; low frequency of reactivity in sporadic ALS cases. Acta Neuropathol Commun 2:55. doi: 10.1186/2051-5960-2-55

5. Basso M, Pozzi S, Tortarolo M, Fiordaliso F, Bisighini C, Pasetto L, Spaltro G, Lidonnici D, Gensano F, Battaglia E et al (2013) Mutant copper-zinc superoxide dismutase (SOD1) induces protein secretion pathway alterations and exosome release in astrocytes: implications for disease spreading and motor neuron pathology in amyotrophic lateral sclerosis. J Biol Chem 288:15699–15711. doi: 10.1074/jbc.M112.425066

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