Notch3 ectodomain is a major component of granular osmiophilic material (GOM) in CADASIL-Reference-Cited by-同舟云学术

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Notch3 ectodomain is a major component of granular osmiophilic material (GOM) in CADASIL

Published:2006-07-27 Issue:3 Volume:112 Page:333-339
ISSN:0001-6322
Container-title:Acta Neuropathologica
language:en
Short-container-title:Acta Neuropathol

Author:

Ishiko Akira,Shimizu Atsushi,Nagata Eiichiro,Takahashi Keikichi,Tabira Takeshi,Suzuki Norihiro

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Pathology and Forensic Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00401-006-0116-2.pdf

Reference29 articles.

1. Arboleda-Velasquez JF, Rampal R, Fung E, Darland DC, Liu M, Martinez MC, Donahue CP, Navarro-Gonzalez MF, Libby P, D’Amore PA, Aikawa M, Haltiwanger RS, Kosik KS (2005) CADASIL mutations impair Notch3 glycosylation by Fringe. Hum Mol Genet 14:1631–1639

2. Artavanis-Tsakonas S, Rand MD, Lake RJ (1999) Notch signaling: cell fate control and signal integration in development. Science 284:770–776

3. Brulin P, Godfraind C, Leteurtre E, Ruchoux MM (2002) Morphometric analysis of ultrastructural vascular changes in CADASIL: analysis of 50 skin biopsy specimens and pathogenic implications. Acta Neuropathol 104:241–248

4. Chabriat H, Vahedi K, Iba-Zizen MT, Joutel A, Nibbio A, Nagy TG, Krebs MO, Julien J, Dubois B, Ducrocq X, Levasseur M, Homeyer P, Mas JL, Lyon-Caen O, Tournier-Lasserve E, Bousser MG (1995) Clinical spectrum of CADASIL: a study of 7 families. Lancet 346:934–939

5. Chabriat H, Levy C, Taillia H, Iba-Zizen MT, Vahedi K, Joutel A, Tournier-Lasserve E, Bousser MG (1998) Patterns of MRI lesions in CADASIL. Neurology 51:452–457

Cited by 107 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Blood vessel organoids generated by base editing and harboring single nucleotide variation in Notch3 effectively recapitulate CADASIL-related pathogenesis;Molecular Neurobiology;2024-04-09

2. Protein aggregates containing wild-type and mutant NOTCH3 are major drivers of arterial pathology in CADASIL;Journal of Clinical Investigation;2024-02-22

3. Blood vessel organoids generated by base editing and harboring single nucleotide variation in Notch3 effectively recapitulate CADASIL-related pathogenesis;2024-01-23

4. Progress to Clarify How NOTCH3 Mutations Lead to CADASIL, a Hereditary Cerebral Small Vessel Disease;Biomolecules;2024-01-18

5. Impaired cerebral interstitial fluid dynamics in cerebral autosomal dominant arteriopathy with subcortical infarcts and leucoencephalopathy;Brain Communications;2023-12-19

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