Loss of TDP-43 splicing repression occurs early in the aging population and is associated with Alzheimer’s disease neuropathologic changes and cognitive decline-Reference-Cited by-同舟云学术

Loss of TDP-43 splicing repression occurs early in the aging population and is associated with Alzheimer’s disease neuropathologic changes and cognitive decline

Published:2023-12-22 Issue:1 Volume:147 Page:
ISSN:0001-6322
Container-title:Acta Neuropathologica
language:en
Short-container-title:Acta Neuropathol

Author:

Chang Koping,Ling Jonathan P.,Redding-Ochoa Javier,An Yang,Li Ling,Dean Stephanie A.,Blanchard Thomas G.,Pylyukh Tatiana,Barrett Alexander,Irwin Katherine E.,Moghekar Abhay,Resnick Susan M.,Wong Philip C.,Troncoso Juan C.

Funder

National Institute on Aging

National Institute of Neurological Disorders and Stroke

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Pathology and Forensic Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00401-023-02653-2.pdf

Reference95 articles.

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3. Arnold SJ, Dugger BN, Beach TG (2013) TDP-43 deposition in prospectively followed, cognitively normal elderly individuals: correlation with argyrophilic grains but not other concomitant pathologies. Acta Neuropathol 126:51–57. https://doi.org/10.1007/s00401-013-1110-0

4. Bailly M, Destrieux C, Hommet C, Mondon K, Cottier JP, Beaufils E et al (2015) Precuneus and cingulate cortex atrophy and hypometabolism in patients with alzheimer’s disease and mild cognitive impairment: MRI and (18)F-FDG PET quantitative analysis using FreeSurfer. Biomed Res Int 2015:583931. https://doi.org/10.1155/2015/583931

5. Bell WR, An Y, Kageyama Y, English C, Rudow GL, Pletnikova O et al (2019) Neuropathologic, genetic, and longitudinal cognitive profiles in primary age-related tauopathy (PART) and Alzheimer’s disease. Alzheimers Dement 15:8–16. https://doi.org/10.1016/j.jalz.2018.07.215

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