Preliminary Research: Application of Non-Invasive Measure of Cytochrome c Oxidase Redox States and Mitochondrial Function in a Porcine Model of Carbon Monoxide Poisoning-Reference-Cited by-同舟云学术

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Preliminary Research: Application of Non-Invasive Measure of Cytochrome c Oxidase Redox States and Mitochondrial Function in a Porcine Model of Carbon Monoxide Poisoning

Published:2022-04-28 Issue:3 Volume:18 Page:214-222
ISSN:1556-9039
Container-title:Journal of Medical Toxicology
language:en
Short-container-title:J. Med. Toxicol.

Author:

Lewis Alistair^ORCID,Forti Rodrigo M.,Alomaja Oladunni,Mesaros Clementina,Piel Sarah,Greenwood John C.,Talebi Fatima M.,Mavroudis Constantine D.,Kelly Matthew,Kao Shih-Han,Shofer Frances S.,Ehinger Johannes K.,Kilbaugh Todd J.,Baker Wesley B.,Jang David H.^ORCID

Funder

National Heart, Lung, and Blood Institute

National Institute of Environmental Health Sciences

National Institute of Neurological Disorders and Stroke

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

https://link.springer.com/content/pdf/10.1007/s13181-022-00892-5.pdf

Reference26 articles.

1. Yoon SS, Macdonald SC, Parrish RG. Deaths from unintentional carbon monoxide poisoning and potential for prevention with carbon monoxide detectors. JAMA. 1998;279(9):685–7. https://doi.org/10.1001/jama.279.9.685.

2. Rose JJ, Wang L, Xu Q, McTiernan CF, Shiva S, Tejero J, et al. Carbon monoxide poisoning: pathogenesis, management, and future directions of therapy. Am J Respir Crit Care Med. 2017;195(5):596–606. https://doi.org/10.1164/rccm.201606-1275CI.

3. Pepe G, Castelli M, Nazerian P, Vanni S, Del Panta M, Gambassi F et al. Delayed neuropsychological sequelae after carbon monoxide poisoning: predictive risk factors in the Emergency Department. A retrospective study. Scand J Trauma Resusc Emerg Med 2011;19:16. https://doi.org/10.1186/1757-7241-19-16.

4. Taskiran D, Kutay FZ, Pogun S. Effect of carbon monoxide on dopamine and glutamate uptake and cGMP levels in rat brain. Neuropsychopharmacology. 2003;28(6):1176–81. https://doi.org/10.1038/sj.npp.1300132.

5. Thom SR. Leukocytes in carbon monoxide-mediated brain oxidative injury. Toxicol Appl Pharmacol. 1993;123(2):234–47. https://doi.org/10.1006/taap.1993.1242.

Cited by 5 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Carbon monoxide as a cellular protective agent in a swine model of cardiac arrest protocol;PLOS ONE;2024-05-15

2. Investigation of Cerebral Mitochondrial Injury in a Porcine Survivor Model of Carbon Monoxide Poisoning;Journal of Medical Toxicology;2023-10-17

3. Cell-Free DNA as a Biomarker in a Rodent Model of Chlorpyrifos Poisoning Causing Mitochondrial Dysfunction;Journal of Medical Toxicology;2023-07-31

4. Alteration in Cerebral Metabolism in a Rodent Model of Acute Sub-lethal Cyanide Poisoning;Journal of Medical Toxicology;2023-02-09

5. Correction to: Preliminary Research: Application of Non-Invasive Measure of Cytochrome c Oxidase Redox States and Mitochondrial Function in a Porcine Model of Carbon Monoxide Poisoning;Journal of Medical Toxicology;2022-12-12

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