Ultrastructural and Functional Alterations of EC Coupling Elements in mdx Cardiomyocytes: An Analysis from Membrane Surface to Depth
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Biochemistry,General Medicine,Biophysics
Link
http://link.springer.com/content/pdf/10.1007/s12013-013-9517-8.pdf
Reference39 articles.
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2. Ervasti, J. M., Kahl, S. D., & Campbell, K. P. (1991). Purification of dystrophin from skeletal muscle. Journal of Biological Chemistry, 266, 9161–9165.
3. Ferlini, A., Sewry, C., Melis, M. A., Mateddu, A., & Muntoni, F. (1999). X-linked dilated cardiomyopathy and the dystrophin gene. Neuromuscular Disorders, 9, 339–346.
4. Bostick, B., Yue, Y., Long, C., & Duan, D. (2008). Prevention of dystrophin-deficient cardiomyopathy in twenty-one-month-old carrier mice by mosaic dystrophin expression or complementary dystrophin/utrophin expression. Circulation Research, 102, 121–130.
5. Quinlan, J. G., Hahn, H. S., Wong, B. L., Lorenz, J. N., Wenisch, A. S., & Levin, L. S. (2004). Evolution of the mdx mouse cardiomyopathy: physiological and morphological findings. Neuromuscular Disorders, 14, 491–496.
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