Progress in Understanding the Role and Therapeutic Targets of Polarized Subtypes of Macrophages in Pulmonary Fibrosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Biochemistry,General Medicine,Biophysics
Link
https://link.springer.com/content/pdf/10.1007/s12013-023-01182-9.pdf
Reference77 articles.
1. Ballester B., Milara J., Cortijo J. (2019). Idiopathic Pulmonary Fibrosis and Lung Cancer: Mechanisms and Molecular Targets. International Journal of Molecular Science, 20. https://doi.org/10.3390/ijms20030593.
2. Maher, T. M., Bendstrup, E., & Dron, L., et al. (2021). Global incidence and prevalence of idiopathic pulmonary fibrosis. Respiratory Research, 22, 197. https://doi.org/10.1186/s12931-021-01791-z.
3. McDonald, L. T. (2021). Healing after COVID-19: are survivors at risk for pulmonary fibrosis? American Journal of Physiology-Lung Cellular and Molecular Physiology, 320, L257–l265. https://doi.org/10.1152/ajplung.00238.2020.
4. George, P. M., Wells, A. U., & Jenkins, R. G. (2020). Pulmonary fibrosis and COVID-19: the potential role for antifibrotic therapy. The Lancet Respiratory Medicine, 8, 807–815. https://doi.org/10.1016/s2213-2600(20)30225-3.
5. Thannickal, V. J., Toews, G. B., White, E. S., Lynch, III, J. P., & Martinez, F. J. (2004). Mechanisms of pulmonary fibrosis. Annual Review of Medicine, 55, 395–417. https://doi.org/10.1146/annurev.med.55.091902.103810.
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