Overexpressed FAM111B degrades GSDMA to promote esophageal cancer tumorigenesis and cisplatin resistance-Reference-Cited by-同舟云学术

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Overexpressed FAM111B degrades GSDMA to promote esophageal cancer tumorigenesis and cisplatin resistance

Published:2023-09-06 Issue: Volume: Page:
ISSN:2211-3428
Container-title:Cellular Oncology
language:en
Short-container-title:Cell Oncol.

Author:

Wang Haiqin,Wang Haohui,Chen Jiajing,Liu Pian,Xiao Xiaoxiong

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Oncology,Molecular Medicine,General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s13402-023-00871-0.pdf

Reference38 articles.

1. H.C. Puhr, G.W. Prager, A. Ilhan-Mutlu, How we treat esophageal squamous cell carcinoma. ESMO Open 8(1), 100789 (2023)

2. M. Sheikh et al., Current status and future prospects for esophageal cancer. Cancers (Basel) 15(3), 765 (2023)

3. J. An et al., Natural products for esophageal cancer therapy: from traditional medicine to modern drug discovery. Int J Mol Sci 23(21), 13558 (2022)

4. Y. Baba et al., Tumor immune microenvironment and immune checkpoint inhibitors in esophageal squamous cell carcinoma. Cancer Sci 111(9), 3132–3141 (2020)

5. Y. Jia et al., Long non-coding RNA NORAD/miR-224-3p/MTDH axis contributes to CDDP resistance of esophageal squamous cell carcinoma by promoting nuclear accumulation of β-catenin. Mol Cancer 20(1), 162 (2021)

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