USP18 promotes the growth in hemangiomas by regulating PI3K/AKT pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health,General Pharmacology, Toxicology and Pharmaceutics,Toxicology,Pathology and Forensic Medicine
Link
https://link.springer.com/content/pdf/10.1007/s13273-021-00146-5.pdf
Reference27 articles.
1. Burkart C et al (2013) Usp18 deficient mammary epithelial cells create an antitumour environment driven by hypersensitivity to IFN-λ and elevated secretion of Cxcl10. EMBO Mol Med 5:1035–1050. https://doi.org/10.1002/emmm.201201864
2. Burks J, Reed RE, Desai SD (2014) ISGylation governs the oncogenic function of Ki-Ras in breast cancer. Oncogene 33:794–803. https://doi.org/10.1038/onc.2012.633
3. Calicchio ML, Collins T, Kozakewich HP (2009) Identification of signaling systems in proliferating and involuting phase infantile hemangiomas by genome-wide transcriptional profiling. Am J Pathol 174:1638–1649. https://doi.org/10.2353/ajpath.2009.080517
4. Chang L et al (2008) Growth characteristics of infantile hemangiomas: implications for management. Pediatrics 122:360–367. https://doi.org/10.1542/peds.2007-2767
5. Diao W et al (2020) USP18 promotes cell proliferation and suppressed apoptosis in cervical cancer cells via activating AKT signaling pathway. BMC Cancer 20:741–741. https://doi.org/10.1186/s12885-020-07241-1
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