NME1 functions as a metastasis suppressor in pancreatic cancer
Author:
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health,General Pharmacology, Toxicology and Pharmaceutics,Toxicology,Pathology and Forensic Medicine
Link
https://link.springer.com/content/pdf/10.1007/s13273-022-00305-2.pdf
Reference28 articles.
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3. Dongre A, Weinberg RA (2019) New insights into the mechanisms of epithelial–mesenchymal transition and implications for cancer. Nat Rev Mol Cell Biol 20(2):69–84
4. Fischer KR, Durrans A, Lee S, Sheng J, Li F, Wong ST, Choi H, El Rayes T, Ryu S, Troeger J (2015) Epithelial-to-mesenchymal transition is not required for lung metastasis but contributes to chemoresistance. Nature 527(7579):472–476
5. Huna A, Nawrocki-Raby B, Padilla-Benavides T, Gavard J, Coscoy S, Bernard D, Boissan M (2021) Loss of the metastasis suppressor NME1, but not of its highly related isoform NME1, induces a hybrid epithelial-mesenchymal state in cancer cells. Int J Mole Sci 22(7):3718
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