Allylpyrocatechol ameliorates sepsis-induced lung injury via SIRT1-mediated suppression of p65 and nucleocytoplasmic translocation of HMGB1-Reference-Cited by-同舟云学术

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Allylpyrocatechol ameliorates sepsis-induced lung injury via SIRT1-mediated suppression of p65 and nucleocytoplasmic translocation of HMGB1

Published:2021-05-27 Issue:4 Volume:17 Page:397-407
ISSN:1738-642X
Container-title:Molecular & Cellular Toxicology
language:en
Short-container-title:Mol. Cell. Toxicol.

Author:

Mu Yanfei,Mu Xiaosong,Yang Yan,Zhou Yanhong

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health,General Pharmacology, Toxicology and Pharmaceutics,Toxicology,Pathology and Forensic Medicine

Link

https://link.springer.com/content/pdf/10.1007/s13273-021-00145-6.pdf

Reference33 articles.

1. Andersson U, Tracey KJ (2011) HMGB1 is a therapeutic target for sterile inflammation and infection. Annu Rev Immunol 29:139–162

2. Angus DC, van der Poll T (2013) Severe sepsis and septic shock. N Engl J Med 369:840–851

3. Bhattacharya S, Banerjee D, Bauri AK, Chattopadhyay S, Bandyopadhyay SK (2007) Healing property of the Piper betel phenol, allylpyrocatechol against indomethacin-induced stomach ulceration and mechanism of action. World J Gastroenterol 13(27):3705–3713

4. Bonaldi T, Talamo F, Scaffidi P, Ferrera D, Porto A, Bachi A, Rubartelli A, Agresti A, Bianchi ME (2003) Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion. EMBO J 22(20):5551–5560

5. Chaudry IH, Wichterman KA, Baue AE (1979) Effect of sepsis on tissue adenine nucleotide levels. Surgery 85(2):205–211

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