Aldosterone-related genetic effects in hypertension
Author:
Publisher
Springer Science and Business Media LLC
Subject
Internal Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11906-000-0013-3.pdf
Reference53 articles.
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3. Shimkets RA, Warnock DG, Bositis CM, et al.: Liddle’s syndrome: heritable human hypertension caused by mutations in the β subunit of the epithelial sodium channel. Cell 1994, 79:407–414.
4. Jeunemaitre X, Bassilana F, Persu A, et al.: Genotype-phenotype analysis of a newly discovered family with Liddle’s syndrome. J Hypertens 1997, 15:1091–1100.
5. Abriel H, Loffing H, Rebhus JF, et al.: Defective regulation of the epithelial Na+ channel by NEDD4 in Liddle’s syndrome. J Clin Invest 1999, 103:667–673. Elegant, definitive studies of the binding interactions between NEDD4 and the ENaC subunits.
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1. Pathophysiology and Treatment of Resistant Hypertension: The Role of Aldosterone and Amiloride-Sensitive Sodium Channels;Seminars in Nephrology;2014-09
2. Direct Activation of ENaC by Angiotensin II: Recent Advances and New Insights;Current Hypertension Reports;2012-11-18
3. Aldosterone-sensitive repression ofENaCαtranscription by a histone H3 lysine-79 methyltransferase;American Journal of Physiology-Cell Physiology;2006-03
4. Epithelial Sodium Channel and the Control of Sodium Balance: Interaction Between Genetic and Environmental Factors;Annual Review of Physiology;2002-03
5. Genetics of Hypertension;Developments in Cardiovascular Medicine;2001
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