Sympathetic nervous system function in renal hypertension
Author:
Publisher
Springer Science and Business Media LLC
Subject
Internal Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11906-002-0012-7.pdf
Reference43 articles.
1. United States Renal Data System: 1999 Annual Data Report. Bethesda: The National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases; 1999.
2. Levey AS: Controlling the epidemic of cardiovascular disease in chronic renal disease: where do we start? Am J Kidney Dis 1998, 32:S5-S13.
3. Cheigh JS, Milite C, Sullivan JF, et al.: Hypertension is not adequately controlled in hemodialysis patients. Am J Kidney Dis 1992, 19:453–459.
4. Salem MM: Hypertension in the hemodialysis population: a survey of 649 patients. Am J Kidney Dis 1995, 26:461–468.
5. Agodoa LY, Appel L, Bakris GL, et al.: Effect of ramipril vs amlodipine on renal outcomes in hypertensive nephrosclerosis: a randomized controlled trial. JAMA 2001, 285:2719–2728. The AASK study compared the effects of stringent blood pressure control with multidrug regimens that included either the ACE inhibitor ramipril or the dihydropyridine CCB amlodipine on the progression of hypertensive nephrosclerosis. In the patients with baseline proteinuria, the progression of renal failure was slower with the ramipril-based regimen. This is the first study to show an advantage of ACE inhibitors in hypertensive African-Americans, an ethnic group in which this class of antihypertensive agent has been thought to be relatively ineffective.
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