A population pharmacokinetic model for individualized regimens of finasteride according to CYP3A5 genotype and liver function
Author:
Funder
Handok Jeseok Foundation
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology, Toxicology and Pharmaceutics (miscellaneous),Pharmaceutical Science
Link
https://link.springer.com/content/pdf/10.1007/s40005-023-00638-7.pdf
Reference32 articles.
1. Almeida A, Almeida S, Filipe A, Gagnon S, Mirapeix A et al (2005) Bioequivalence study of two different coated tablet formulations of finasteride in healthy volunteers. Arzneimittelforschung 55(4):218–222. https://doi.org/10.1055/s-0031-1296848
2. Chau CH, Price DK, Till C, Goodman PJ, Chen X et al (2015) Finasteride concentrations and Prostate cancer risk: results from the Prostate Cancer Prevention Trial. PLoS ONE 10(5):e0126672. https://doi.org/10.1371/journal.pone.0126672
3. Chen L, Jiang X, Huang L, Lan K, Wang H et al (2009) Bioequivalence of a single 10-mg dose of finasteride 5-mg oral disintegrating tablets and standard tablets in healthy adult male Han Chinese volunteers: a randomized sequence, open-label, two-way crossover study. Clin Ther 31(10):2242–2248. https://doi.org/10.1016/j.clinthera.2009.09.015
4. Cilotti A, Danza G, Serio M (2001) Clinical application of 5α-reductase inhibitors. J Endocrinol Invest 24(3):199–203. https://doi.org/10.1007/BF03343844
5. Diviccaro S, Melcangi RC, Giatti S (2019) Post-finasteride syndrome: an emerging clinical problem. Neurobiol Stress 12:100209. https://doi.org/10.1016/j.ynstr.2019.100209
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