DDX17: A Potential Target for Heart Failure Therapies
Author:
Funder
Science and Technology Commission of Shanghai Municipality
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s12265-024-10549-z.pdf
Reference5 articles.
1. Fuller-Pace FV. DExD/H box RNA helicases: multifunctional proteins with important roles in transcriptional regulation. Nucleic Acids Res. 2006;34(15):4206–15. https://doi.org/10.1093/nar/gkl460.
2. Lin B, Wang F, Wang J, et al. The protective role of p72 in doxorubicin-induced cardiomyocytes injury in vitro. Mol Med Rep. 2016;14(4):3376–80. https://doi.org/10.3892/mmr.2016.5600.
3. Gao R, Wang L, Bei Y, et al. Long noncoding RNA cardiac physiological hypertrophy-associated regulator induces cardiac physiological hypertrophy and promotes functional recovery after myocardial ischemia-reperfusion injury. Circulation. 2021;144(4):303–17. https://doi.org/10.1161/circulationaha.120.050446.
4. Yan M, Gao J, Lan M, et al. DEAD-box helicase 17 (DDX17) protects cardiac function by promoting mitochondrial homeostasis in heart failure. Signal Transduct Target Ther. 2024;9(1):127. https://doi.org/10.1038/s41392-024-01831-2.
5. Raffin J, Rolland Y, Genoux A, et al. Associations between physical activity levels and ATPase inhibitory factor 1 concentrations in older adults. J Sport Health Sci. 2024;13(3):409–18. https://doi.org/10.1016/j.jshs.2023.09.009.
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