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The Genome-Wide Association Study—A New Era for Common Polygenic Disorders

Published:2010-03-27 Issue:3 Volume:3 Page:173-182
ISSN:1937-5387
Container-title:Journal of Cardiovascular Translational Research
language:en
Short-container-title:J. of Cardiovasc. Trans. Res.

Author:

Roberts Robert,Wells George A.,Stewart Alexandre F. R.,Dandona Sonny,Chen Li

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Cardiology and Cardiovascular Medicine,Pharmaceutical Science,Genetics,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1007/s12265-010-9178-6.pdf

Reference70 articles.

1. Redon, R., Ishikawa, S., Fitch, L., et al. (2006). Global variation in copy number in the human genome. Nature, 444(7118), 444–454.

2. McCarroll, S. (2008). Extending genome-wide association studies to copy-number variation. Human Molecular Genetics, 17(R2), R135–R142.

3. The International HapMap Consortium. (2003). The international HapMap project. Science, 426, 789–796.

4. Tregouet, D. A., et al. (2009). Genome-wide haplotype association study identifies the SLC22A3–LPAL2–LPA gene cluster as a risk locus for Coronaroy Artery Disease. Nature Genetics, 41, 283–285.

5. Collins, F. S., & McKusick, V. A. (2001). Implications of the Human Genome Project for medical science. JAMA, 285(5), 540–544.

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