CAV1 Protein Encapsulated in Mouse BMSC-Derived Extracellular Vesicles Alleviates Myocardial Fibrosis Following Myocardial Infarction by Blocking the TGF-β1/SMAD2/c-JUN Axis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics (clinical),Cardiology and Cardiovascular Medicine,Pharmaceutical Science,Genetics,Molecular Medicine
Link
https://link.springer.com/content/pdf/10.1007/s12265-023-10472-9.pdf
Reference63 articles.
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2. Zhao Z, Du S, Shen S, Wang L. microRNA-132 inhibits cardiomyocyte apoptosis and myocardial remodeling in myocardial infarction by targeting IL-1beta. J Cell Physiol. 2020;235:2710–21. https://doi.org/10.1002/jcp.29175.
3. Golforoush P, Yellon DM, Davidson SM. Mouse models of atherosclerosis and their suitability for the study of myocardial infarction. Basic Res Cardiol. 2020;115:73. https://doi.org/10.1007/s00395-020-00829-5.
4. Daseke MJ 2nd, Tenkorang MAA, Chalise U, Konfrst SR, Lindsey ML. Cardiac fibroblast activation during myocardial infarction wound healing: fibroblast polarization after MI. Matrix Biol. 2020;91-92:109–16. https://doi.org/10.1016/j.matbio.2020.03.010.
5. Kurose H. Cardiac Fibrosis and Fibroblasts. Cells. 2021;10 https://doi.org/10.3390/cells10071716.
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