AAV8-Mediated Long-Term Expression of Human LCAT Significantly Improves Lipid Profiles in hCETP;Ldlr+/− Mice-Reference-Cited by-同舟云学术

AAV8-Mediated Long-Term Expression of Human LCAT Significantly Improves Lipid Profiles in hCETP;Ldlr+/− Mice

Published:2011-08-06 Issue:6 Volume:4 Page:801-810
ISSN:1937-5387
Container-title:Journal of Cardiovascular Translational Research
language:en
Short-container-title:J. of Cardiovasc. Trans. Res.

Author:

Chen Zhu,Chu Donald,Castro-Perez Jose M.,Ni Weihua,Zhang Aiwu,Krsmanovic Mihajlo L.,Xie Dan,Shah Vinit,Stout Steven J.,McLaren David G.,Stefanni Alice C.,Lee Sang Ho,Roddy Thomas P.,Plump Andrew S.,Hubbard Brian K.,Vogt Thomas F.,Zhou Heather H.

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Cardiology and Cardiovascular Medicine,Pharmaceutical Science,Genetics,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1007/s12265-011-9309-8.pdf

Reference42 articles.

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3. Francone, O. L., Gurakar, A., & Fielding, C. (1989). Distribution and functions of lecithin:cholesterol acyltransferase and cholesteryl ester transfer protein in plasma lipoproteins. Evidence for a functional unit containing these activities together with apolipoproteins A-I and D that catalyzes the esterification and transfer of cell-derived cholesterol. Journal of Biological Chemistry, 264, 7066–7072.

4. Rader, D. J., Ikewaki, K., Duverger, N., Schmidt, H., Pritchard, H., Frohlich, J., et al. (1994). Markedly accelerated catabolism of apolipoprotein A-II (ApoA-II) and high density lipoproteins containing ApoA-II in classic lecithin: cholesterol acyltransferase deficiency and fish-eye disease. The Journal of Clinical Investigation, 93, 321–330.

5. Fielding, C. J., & Fielding, P. E. (1995). Molecular physiology of reverse cholesterol transport. Journal of Lipid Research, 36, 211–228.

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